APC mutations in colorectal tumours from FAP patients are selected for CtBP-mediated oligomerization of truncated APC

By Schneikert Jean   Brauburger Katharina   Behrens Jürgen  

Human Molecular Genetics, Vol. 20, Iss. 18, 2011-09 ,pp. : 3554-3564

Oxford University Press

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-Catenin degradation mediated by the CID domain of APC provides a model for the selection of APC mutations in colorectal, desmoid and duodenal tumours

By Kohler Eva Maria   Chandra Shree Harsha Vijaya   Behrens Jrgen   Schneikert Jean  

Human Molecular Genetics, Vol. 18, Iss. 2, 2009-01 ,pp. : 213-226

Oxford University Press

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The ‘just-right’ signaling model: APC somatic mutations are selected based on a specific level of activation of the  -catenin signaling cascade

By Albuquerque Cristina  

Human Molecular Genetics, Vol. 11, Iss. 13, 2002-06 ,pp. : 1549-1560

Oxford University Press

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Familial Infiltrative Fibromatosis (Desmoid Tumours) (MIM135290) Caused by a Recurrent 3′ APC Gene Mutation

By Scott Rodney J.   Froggatt Nicola J.   Trembath Richard C.   Evans D. Gareth R.   Hodgson Shirley V.   Maher Eamonn R.  

Human Molecular Genetics, Vol. 5, Iss. 12, 1996-12 ,pp. : 1921-1924

Oxford University Press

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Primary non-random X inactivation associated with disruption of Xist promoter regulation

By Newall A.E.T.  

Human Molecular Genetics, Vol. 10, Iss. 6, 2001-03 ,pp. : 581-589

Oxford University Press

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