Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy

Author： Wehling-Henricks Michelle   Oltmann Meredith   Rinaldi Chiara   Myung Kyu H.   Tidball James G.  

Publisher： Oxford University Press

ISSN： 1460-2083

Source： Human Molecular Genetics, Vol.18, Iss.18, 2009-09, pp. : 3439-3451

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Loss of Aggression, After Transfer onto a C57BL/6J Background, in Mice Carrying a Targeted Disruption of the Neuronal Nitric Oxide Synthase Gene

By Le Roy I.  

Behavior Genetics, Vol. 30, Iss. 5, 2000-09 ,pp. : 367-373

Springer Publishing Company

Access to resources Recommend Favorite

Glycosylation defects: a new mechanism for muscular dystrophy?

By Grewal Prabhjit K.   Hewitt Jane E.  

Human Molecular Genetics, Vol. 12, Iss. 2, 2003-10 ,pp. : 259-264

Oxford University Press

Access to resources Recommend Favorite

Cardiomyopathy in dystrophin-deficient hearts is prevented by expression of a neuronal nitric oxide synthase transgene in the myocardium

By Wehling-Henricks Michelle  

Human Molecular Genetics, Vol. 14, Iss. 14, 2005-07 ,pp. : 1921-1933

Oxford University Press

Access to resources Recommend Favorite

Simple Tandem Repeat Polymorphisms in the Neuronal Nitric Oxide Synthase Gene in Different Ethnic Populations

By  

Human Heredity, Vol. 49, Iss. 3, 1999-05 ,pp. : 139-141

Karger

Access to resources Recommend Favorite

Overexpression of P104L mutant caveolin-3 in mice develops hypertrophic cardiomyopathy with enhanced contractility in association with increased endothelial nitric oxide synthase activity

By Ohsawa Yutaka  

Human Molecular Genetics, Vol. 13, Iss. 2, 2004-01 ,pp. : 151-157

Oxford University Press

Access to resources Recommend Favorite