Oxidative stress causes abnormal accumulation of familial amyotrophic lateral sclerosis-related mutant SOD1 in transgenic Caenorhabditis elegans

By Ishii N.   Oeda T.   Shimohama S.   Kitagawa N.   Kohno R.   Imura T.   Shibasaki H.  

Human Molecular Genetics, Vol. 10, Iss. 19, 2001-09 ,pp. : 2013-2023

Oxford University Press

Access to resources Recommend Favorite

SOD1 and TDP-43 animal models of amyotrophic lateral sclerosis: recent advances in understanding disease toward the development of clinical treatments

By Joyce Peter  

Mammalian Genome, Vol. 22, Iss. 7-8, 2011-08 ,pp. : 420-448

Springer Publishing Company

Access to resources Recommend Favorite

Calcium-permeable AMPA receptors promote misfolding of mutant SOD1 protein and development of amyotrophic lateral sclerosis in a transgenic mouse model

By Tateno Minako  

Human Molecular Genetics, Vol. 13, Iss. 19, 2004-10 ,pp. : 2183-2196

Oxford University Press

Access to resources Recommend Favorite

A novel mouse model with impaired dynein/dynactin function develops amyotrophic lateral sclerosis (ALS)-like features in motor neurons and improves lifespan in SOD1-ALS mice

By Teuling Eva   van Dis Vera   Wulf Phebe S.   Haasdijk Elize D.   Akhmanova Anna   Hoogenraad Casper C.   Jaarsma Dick  

Human Molecular Genetics, Vol. 17, Iss. 18, 2008-09 ,pp. : 2849-2862

Oxford University Press

Access to resources Recommend Favorite

NIPA1 polyalanine repeat expansions are associated with amyotrophic lateral sclerosis

By Blauw Hylke M.   van Rheenen Wouter   Koppers Max   Van Damme Philip   Waibel Stefan   Lemmens Robin   van Vught Paul W. J.   Meyer Thomas   Schulte Claudia   Gasser Thomas   Cuppen Edwin   Pasterkamp R. Jeroen   Robberecht Wim   Ludolph Albert C.   Veldink Jan H.   van den Berg Leonard H.  

Human Molecular Genetics, Vol. 21, Iss. 11, 2012-06 ,pp. : 2497-2502

Oxford University Press

Access to resources Recommend Favorite