Suppression of polyglutamine-induced protein aggregation in Caenorhabditis elegans by torsin proteins

By Caldwell G.A.   Cao S.   Sexton E.G.   Gelwix C.C.   Bevel J.P.   Caldwell K.A.  

Human Molecular Genetics, Vol. 12, Iss. 3, 2003-02 ,pp. : 307-319

Oxford University Press

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Neuronal dysfunction in a polyglutamine disease model occurs in the absence of ubiquitin–proteasome system impairment and inversely correlates with the degree of nuclear inclusion formation

By Bowman Aaron B.  

Human Molecular Genetics, Vol. 14, Iss. 5, 2005-03 ,pp. : 679-691

Oxford University Press

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Suppression of polyglutamine-induced toxicity in cell and animal models of Huntington's disease by ubiquilin

By Wang Hongmin   Lim Precious J.   Vogel Bruce E.   Monteiro Mervyn J.  

Human Molecular Genetics, Vol. 15, Iss. 6, 2006-03 ,pp. : 1025-1041

Oxford University Press

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Expanded polyglutamine domain possesses nuclear export activity which modulates subcellular localization and toxicity of polyQ disease protein via exportin-1

By Chan Wing Man   Tsoi Ho   Wu Chi Chung   Wong Chi Hang   Cheng Tat Cheung   Li Hoi Yeung   Lau Kwok Fai   Shaw Pang Chui   Perrimon Norbert   Chan Ho Yin Edwin  

Human Molecular Genetics, Vol. 20, Iss. 9, 2011-05 ,pp. : 1738-1750

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Pathogenesis of polyglutamine disorders: aggregation revisited

By Michalik Andrej   Van Broeckhoven Christine  

Human Molecular Genetics, Vol. 12, Iss. 2, 2003-10 ,pp. : 173-186

Oxford University Press

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