Population and Computational Analysis of the MGEA6 P521A Variation as a Risk Factor for Familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease)

By Lemos Roberta   Oliveira Danyllo   Zatz Mayana   Oliveira João  

Journal of Molecular Neuroscience, Vol. 43, Iss. 3, 2011-03 ,pp. : 333-336

Humana Press, Inc

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Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification (“Fahr’s Disease”)

By Oliveira J.   Sobrido M.   Spiteri E.   Hopfer S.   Meroni G.   Petek E.   Baquero M.   Geschwind D.  

Journal of Molecular Neuroscience, Vol. 33, Iss. 2, 2007-10 ,pp. : 151-154

Humana Press, Inc

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Fahr’s disease: variable presentations in a family

By Ashtari Fereshteh   Fatehi Farzad  

Neurological Sciences, Vol. 31, Iss. 5, 2010-10 ,pp. : 665-667

Springer Publishing Company

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Loss of Function of Slc20a2 Associated with Familial Idiopathic Basal Ganglia Calcification in Humans Causes Brain Calcifications in Mice

By Jensen Nina   Schrøder Henrik   Hejbøl Eva   Füchtbauer Ernst-Martin   Oliveira João   Pedersen Lene  

Journal of Molecular Neuroscience, Vol. 51, Iss. 3, 2013-11 ,pp. : 994-999

Humana Press, Inc

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Brain structural and functional abnormalities in Fahr’s disease : a report of two cases

By Messina Roberta   Rocca Maria   Colombo Bruno   Falini Andrea   Comi Giancarlo   Filippi Massimo  

Journal of Neurology, Vol. 260, Iss. 7, 2013-07 ,pp. : 1927-1930

Springer Publishing Company

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