Alteration of Intracellular Na+ During Ischemia in Diabetic Rat Hearts: The Role of Reduced Activity in Na+/H+ Exchange Against Stunning

By Imahashi K.   Hashimoto K.   Yamaguchi H.   Nishimura T.   Kusuoka H.  

Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 3, 1998-03 ,pp. : 509-517

Academic Press

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Decrease in Ischemic Tolerance with Aging in Isolated Perfused Fischer 344 Rat Hearts: Relation to Increases in Intracellular Na+ After Ischemia

By Tani M.   Suganuma Y.   Hasegawa H.   Shinmura K.   Ebihara Y.   Hayashi Y.   Guo X.D.   Takayama M.  

Journal of Molecular and Cellular Cardiology, Vol. 29, Iss. 11, 1997-11 ,pp. : 3081-3089

Academic Press

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Both Na+-K+ ATPase and Na+-H+ Exchanger are Immediately Active upon Post-ischemic Reperfusion in Isolated Rat Hearts

By van Emous J.G.   Schreur J.H.M.   Ruigrok T.J.C.   van Echteld C.J.A.  

Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 2, 1998-02 ,pp. : 337-348

Academic Press

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Ischemic Preconditioning: Effects on pH, Na and Ca in Newborn Rabbit Hearts During Ischemia/Reperfusion

By Liu H.   Cala P.M.   Anderson S.E.  

Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 3, 1998-03 ,pp. : 685-697

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The Sting of Salt on an Old, but Open, Wound—is Na⁺ the Cause of Mitochondrial and Myocardial Injury During Ischemia/Reperfusion?

By Sawyer D.B.   Suter T.M.   Apstein C.S.  

Journal of Molecular and Cellular Cardiology, Vol. 34, Iss. 7, 2002-07 ,pp. : 699-702

Academic Press

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