Myocardial Function in the Working Mouse Heart Overexpressing Cardiac A 1 Adenosine Receptors

By Gauthier N.S.   Headrick J.P.   Paul Matherne G.  

Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 1, 1998-01 ,pp. : 187-193

Academic Press

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Targeted Deletion of the A₃Adenosine Receptor Confers Resistance to Myocardial Ischemic Injury and does not Prevent Early Preconditioning

By Guo Y.   Bolli R.   Bao W.   Wu W-J.   Black Jr R.G.   Murphree S.S.   Salvatore C.A.   Jacobson M.A.   Auchampach J.A.  

Journal of Molecular and Cellular Cardiology, Vol. 33, Iss. 4, 2001-04 ,pp. : 825-830

Academic Press

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Expression of Active _1B-Adrenergic Receptors in the Heart does not Alleviate Ischemic Reperfusion Injury

By Gao X.M.   Wang B.H.   Woodcock E.   Du X.J.  

Journal of Molecular and Cellular Cardiology, Vol. 32, Iss. 9, 2000-09 ,pp. : 1679-1686

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Neovascularization in the human heart is associated with expression of VEGF-A and its receptors Flt-1 (VEGFR-1) and KDR (VEGFR-2). Results from cardiomyopexy in ischemic cardiomyopathy

By Waltenberger J.  

Angiogenesis, Vol. 3, Iss. 4, 1999-01 ,pp. : 345-351

Springer Publishing Company

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Protection Against Myocardial Ischemic/Reperfusion Injury by Inhibitors of Two Separate Pathways of Na+ Entry

By Eng S.   Maddaford T.G.   Kardami E.   Pierce G.N.  

Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 4, 1998-04 ,pp. : 829-835

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