The Role of SR Ca²⁺-Content in Blunted Inotropic Responsiveness of Failing Human Myocardium

By Maier L.S.   Braunhälter J.   Horn W.   Weichert S.   Pieske B.  

Journal of Molecular and Cellular Cardiology, Vol. 34, Iss. 4, 2002-04 ,pp. : 455-467

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Reduced Ca²⁺-Sensitivity of SERCA 2a in Failing Human Myocardium due to Reduced Serin-16 Phospholamban Phoshorylation

By Schwinger R.H.G.   Münch G.   Bölck B.   Karczewski P.   Krause E-G.   Erdmann E.  

Journal of Molecular and Cellular Cardiology, Vol. 31, Iss. 3, 1999-03 ,pp. : 479-491

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L-Type Ca²⁺Channel _1cSubunit Isoform Switching in Failing Human Ventricular Myocardium

By Yang Y.   Chen X.   Margulies K.   Jeevanandam V.   Pollack P.   Bailey B.A.   Houser S.R.  

Journal of Molecular and Cellular Cardiology, Vol. 32, Iss. 6, 2000-06 ,pp. : 973-984

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Frequency-dependent Increase in Cardiac Ca²⁺Current is due to Reduced Ca²⁺Release by the Sarcoplasmic Reticulum

By delgado C.   Artiles A.   Gómez A.M.   Vassort G.  

Journal of Molecular and Cellular Cardiology, Vol. 31, Iss. 10, 1999-10 ,pp. : 1783-1793

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Regional Expression and Functional Characterization of the L-type Ca²⁺-Channel in Myocardium from Patients with End-stage Heart Failure and in Non-failing Human Hearts

By Schwinger R.H.G.   Hoischen S.   Reuter H.   Hullin R.  

Journal of Molecular and Cellular Cardiology, Vol. 31, Iss. 1, 1999-01 ,pp. : 283-296

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