Functional Suppression of Sodium Channels by &bgr;₁-Subunits as a Molecular Mechanism of Idiopathic Ventricular Fibrillation

By Wan X.   Wang Q.   Kirsch G.E.  

Journal of Molecular and Cellular Cardiology, Vol. 32, Iss. 10, 2000-10 ,pp. : 1873-1884

Academic Press

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Pharmacological Characterization of Effects of UK-1745, a Novel Cardiotonic Agent with &bgr; -Adrenoceptor-blocking Action, in Aequorin-loaded Canine Right Ventricular Muscle

By Sawada H.   Endoh M.  

Journal of Molecular and Cellular Cardiology, Vol. 31, Iss. 5, 1999-05 ,pp. : 1047-1062

Academic Press

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CTGF Expression is Induced by TGF- &bgr; in Cardiac Fibroblasts and Cardiac Myocytes: a Potential Role in Heart Fibrosis

By Chen M.M.   Lam A.   Abraham J.A.   Schreiner G.F.   Joly A.H.  

Journal of Molecular and Cellular Cardiology, Vol. 32, Iss. 10, 2000-10 ,pp. : 1805-1819

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&bgr; -Adrenergic Cardiac Hypertrophy is Mediated Primarily by the &bgr;₁-Subtype in the Rat Heart

By Morisco C.   Zebrowski D.C.   Vatner D.E.   Vatner S.F.   Sadoshima J.  

Journal of Molecular and Cellular Cardiology, Vol. 33, Iss. 3, 2001-03 ,pp. : 561-573

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Desensitization of cardiac &bgr; -adrenoceptor signaling with heart failure produced by myocardial infarction in the rat. Evidence for the role of Gi but not Gs or phosphorylating proteins

By Kompa A.R.   Gu X-H.   Evans B.A.   Summers R.J.  

Journal of Molecular and Cellular Cardiology, Vol. 31, Iss. 6, 1999-06 ,pp. : 1185-1201

Academic Press

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