Glaucoma: An Open-Window to Neurodegeneration and Neuroprotection :AN OPEN-WINDOW TO NEURODEGENERATION AND NEUROPROTECTION ( Volume 173 )

Publication subTitle :AN OPEN-WINDOW TO NEURODEGENERATION AND NEUROPROTECTION

Publication series :Volume 173

Author: Nucci   Carlo;Cerulli   Luciano;Osborne   Neville N.  

Publisher: Elsevier Science‎

Publication year: 2008

E-ISBN: 9780080932804

P-ISBN(Paperback): 9780444532565

P-ISBN(Hardback):  9780444532565

Subject: R775 Intraocular pressure and glaucoma.

Language: ENG

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Description

Glaucoma is one of the main causes of blindness throughout the world. It is characterized by death of the retinal ganglion cells, which is associated with loss of the axons making up the optic nerve. Recent studies have demonstrated support for the classification of glaucoma as a degenerative disease of the central nervous system (CNS), leading researchers to look at identifying neuroprotection strategies for the treatment of this disease, like those used for other degenerative diseases of the CNS.

This book will provide an in-depth examination of the most recent findings regarding glaucoma, including risk factors, diagnosis, clinical monitoring, treatment, and above all, the need for treatment based on the concept of neuroprotection. A large part of the book is devoted to research related to this new approach to the treatment of glaucoma.

* Describes the most recent developments on neuroprotection of the optic nerve, including experimental models now used and clinical protocols
* Discusses new strategies for the prevention of neuronal injury in glaucoma patients
* Focuses on evidence-based risk factors, innovative diagnostic aspects and advanced medical/surgical treatment of glaucoma

Chapter

Front cover

pp.:  1 – 4

Copyright page

pp.:  5 – 6

List of Contributors

pp.:  6 – 12

Preface

pp.:  12 – 14

Contents

pp.:  14 – 18

Section II. Anatomical and Functional Monitoring

pp.:  92 – 196

Section III. Current Therapy

pp.:  196 – 294

Section IV. Experimental Approaches to Model Disease

pp.:  294 – 354

Section V. Neuroprotection: New Vistas in Pathophysiology

pp.:  354 – 510

Section VI. Neuroprotection: Evidence for Future Strategies

pp.:  510 – 608

Subject Index

pp.:  608 – 640

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