Pathology-related substitutions in human mitochondrial tRNA Ile reduce precursor 3′ end processing efficiency in vitro

By Levinger L.   Giegé R.   Florentz C.  

Nucleic Acids Research, Vol. 31, Iss. 7, 2003-04 ,pp. : 1904-1912

Oxford University Press

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In vitro 3′-end endonucleolytic processing defect in a human mitochondrial tRNA Ser(UCN) precursor with the U7445C substitution, which causes non-syndromic deafness

By Levinger L.   Jacobs O.   James M.  

Nucleic Acids Research, Vol. 29, Iss. 21, 2001-11 ,pp. : 4334-4340

Oxford University Press

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Processing of Human Mitochondrial tRNA Ser(AGY) GCU : A Novel Pathway in tRNA Biosynthesis

By Rossmanith W.  

Journal of Molecular Biology, Vol. 265, Iss. 4, 1997-04 ,pp. : 365-371

Academic Press

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Impact of disease-related mitochondrial mutations on tRNA structure and function

By Wittenhagen L.M.   Kelley S.O.  

Trends in Biochemical Sciences, Vol. 28, Iss. 11, 2003-11 ,pp. : 605-611

Elsevier

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Characterization of Human Mitochondrial RNase P: Novel Aspects in tRNA Processing

By Rossmanith W.   Karwan R.M.  

Biochemical and Biophysical Research Communications, Vol. 247, Iss. 2, 1998-06 ,pp. : 234-241

Elsevier

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