Light chain-deficient mice produce novel multimeric heavy-chain-only IgA by faulty class switching

By Matheson Louise S.   Osborn Michael J.   Smith Jennifer A.   Corcos Daniel   Hamon Maureen   Chaouaf Rima   Coadwell John   Morgan Geoff   Oxley David   Brggemann Marianne  

International Immunology, Vol. 21, Iss. 8, 2009-08 ,pp. : 957-966

Oxford University Press

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Partial reconstitution of V(D)J rearrangement and lymphocyte development in RAG-deficient mice expressing inducible, tetracycline-regulated RAG transgenes

By Shockett P.E.   Zhou S.   Hong X.   Schatz D.G.  

Molecular Immunology, Vol. 40, Iss. 11, 2004-01 ,pp. : 813-829

Elsevier

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Regulation of V β germline transcription in RAG-deficient mice by the CD3ε-mediated signals: implication of V β transcriptional regulation in TCR β allelic exclusion

By Shinkai Y.   Senoo M.  

International Immunology, Vol. 10, Iss. 5, 1998-05 ,pp. : 553-560

Oxford University Press

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IgA production in MHC class II-deficient mice is primarily a function of B-1a cells

By Snider D.  

International Immunology, Vol. 11, Iss. 2, 1999-02 ,pp. : 191-198

Oxford University Press

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The Ig heavy chain intronic enhancer core region is necessary and sufficient to promote efficient class switch recombination

By Sakai E.  

International Immunology, Vol. 11, Iss. 10, 1999-10 ,pp. : 1709-1713

Oxford University Press

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