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Trends in Cardiovascular Medicine, Vol. 10, Iss. 1, 2000-01 ,pp. : 19-22

Elsevier

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Activation of p38 MAPK Induced by a Multi-cycle Ischaemic Preconditioning Protocol is Associated with Attenuated p38 MAPK Activity During Sustained Ischaemia and Reperfusion

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Decreased p38 MAPK Activity in End-Stage Failing Human Myocardium: p38 MAPK  is the Predominant Isoform Expressed in Human Heart

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Anisomycin Does Not Activate p38 MAPK in Human Platelets - p38 (MAP kinase) activation and protein synthesis inhibition in mammalian cells

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Tumor necrosis factor-induced protection of the murine heart is independent of p38-MAPK activation

By Tanno M.   Gorog D.A.   Bellahcene M.   Cao X.   Quinlan R.A.   Marber M.S.  

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