Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice

By Wong Margaret   Martin Lee J.  

Human Molecular Genetics, Vol. 19, Iss. 11, 2010-06 ,pp. : 2284-2302

Oxford University Press

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Rai1 deficiency in mice causes learning impairment and motor dysfunction, whereas Rai1 heterozygous mice display minimal behavioral phenotypes

By Bi Weimin   Yan Jiong   Shi Xin   Yuva-Paylor Lisa A.   Antalffy Barbara A.   Goldman Alica   Yoo Jong W.   Noebels Jeffrey L.   Armstrong Dawna L.   Paylor Richard   Lupski James R.  

Human Molecular Genetics, Vol. 16, Iss. 15, 2007-08 ,pp. : 1802-1813

Oxford University Press

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Overexpression of Nd1, a novel Kelch family protein, in the heart of transgenic mice protects against doxorubicin-induced cardiomyopathy

By Matsudo Yuji   Takamori Yasuyuki   Fujimura Lisa   Nishio Saori   Sasagawa Kazushi   Komuro Issei   Tokuhisa Takeshi   Hatano Masahiko  

Transgenic Research, Vol. 15, Iss. 5, 2006-10 ,pp. : 573-581

Springer Publishing Company

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Transgenic expression of CECR1 adenosine deaminase in mice results in abnormal development of heart and kidney

By Riazi Ali  

Transgenic Research, Vol. 14, Iss. 3, 2005-06 ,pp. : 333-336

Springer Publishing Company

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Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common feature

By Wang Jiou  

Human Molecular Genetics, Vol. 12, Iss. 21, 2003-11 ,pp. : 2753-2764

Oxford University Press

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