Amyloid-β Decreases Nitric Oxide Production in Cultured Retinal Neurons: A Possible Mechanism for Synaptic Dysfunction in Alzheimer’s Disease?

By Oliveira Leandro   Louzada Paulo   Mello Fernando   Ferreira Sérgio  

Neurochemical Research, Vol. 36, Iss. 1, 2011-01 ,pp. : 163-169

Springer Publishing Company

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Protective effect of ginsenoside Rb1 on β-amyloid protein(1-42)-induced neurotoxicity in cortical neurons

By Qian Yi-Hua   Han Hua   Hu Xiao-Dan   Shi Li-Li  

Neurological Research, Vol. 31, Iss. 7, 2009-09 ,pp. : 663-667

Maney Publishing

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Homocysteine is a risk factor for cerebral small vessel disease, acting via endothelial dysfunction

By Hassan Ahamad  

Brain, Vol. 127, Iss. 1, 2004-01 ,pp. : 212-219

Oxford University Press

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β-Amyloid-induced reactive astrocytes display altered ability to support dendrite and axon growth from mouse cerebral cortical neurons in vitro

By Monnerie Hubert   Esquenazi Susana   Shashidhara Shalini   Le Roux Peter D.  

Neurological Research, Vol. 27, Iss. 5, 2005-07 ,pp. : 525-532

Maney Publishing

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-Amyloid Plaques Induce Neuritic Dystrophy of Nitric Oxide-Producing Neurons in a Transgenic Mouse Model of Alzheimer's Disease

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Experimental Neurology, Vol. 168, Iss. 2, 2001-04 ,pp. : 203-212

Elsevier

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