Description
Neurogenic Heart Lesions reviews the main publications concerned with the problem of neurogenic lesions of the heart. The book describes the methods for producing experimental neurogenic lesions, as well as the pharmacological analysis of the participation of the sympathetic nervous system and its transmitters in the development of neurogenic lesions of the myocardium. The text also discusses the changes in energy metabolism and in functional conditions of the myocardium caused by neurogenic lesions; and the pharmacoprophylaxis and pharmacotherapy of the biochemical changes produced by neurogenic lesions of the myocardium. Pharmacologists, pathologists, and other clinicians and physicians concerned with the new achievements in the field of cardiology will find the book invaluable.
Chapter
Chapter 1. Methods for Producing Experimental Neurogenic Lesions
STRUCTURAL MYOCARDIAL LESIONS PRODUCED BY ELECTRICAL STIMULATION OF THE HYPOTHALAMUS
ULTRASTRUCTURAL AND MICROSCOPIC CHANGES PRODUCED BY REFLEX LESIONS OF THE MYOCARDIUM
PHARMACOLOGICAL AND PHYSIOLOGICAL ANALYSIS OF THE PROPAGATION PATHS OF THE IMPULSES PRODUCING NEUROGENIC LESIONS OF THE HEART
Chapter 2. Pharmacological Analysis of the Participation of the Sympathetic Nervous System and its Transmitters in the Development of Neurogenic Lesions of the Myocardium
NORADRENALINE LEVELS IN MYOCARDIAL TISSUE DURING EXCESSIVE STIMULATION
EXPERIMENTS WITH THE PRECURSOR OF NORADRENALINE – L-DOPA
EXPERIMENTS WITH THE CENTRAL α-ADRENOBLOCKER IEM-611
EXPERIMENTS WITH ETHYMISOL
EXPERIMENTS WITH THE GANGLIOBLOCKER BENZOHEXAMETHONIUM
EXPERIMENTS WITH THE β-BLOCKER PROPRANOLOL
EXPERIMENTS WITH TOXIC DOSES OF CATECHOLAMINES
Chapter 3. Changes in Energy Metabolism and in Functional Conditions of the Myocardium caused by Neurogenic Lesions
GLYCOGEN LEVELS AND THE PROCESS OF GLYCOLYSIS DURING EXCESSIVE STIMULATION OF THE ANIMALS
OXIDATIVE PHOSPHORYLATION IN MITOCHONDRIA OF THE HEART PRODUCED BY ITS NEUROGENIC LESION AND THE INJECTION OF TOXIC DOSES OF NORADRENALINE
HIGH-ENERGY ADENOSINE TRIPHOSPHATE (ATP), CREATINE PHOSPHATE (CP) AND INORGANIC PHOSPHORUS (IP) LEVELS IN THE MYOCARDIUM PRODUCED BY EXCESSIVE STIMULATION
THE ACTIVITY OF THE FIRST STAGE OF THE PENTOSE-PHOSPHATE PATHWAY, GLUCOSE-6-PHOSPHATE DEHYDROGENASE (G-6-P DH)
ELECTROCARDIOGRAPHIC DATA OBTAINED DURING NEUROGENIC LESIONS OF THE MYOCARDIUM AND INJECTION OF LARGE DOSES OF NORADRENALINE
Chapter 4. Pharmacoprophylaxis and Pharmacotherapy of the Biochemical Changes Produced by Neurogenic Lesions of the Myocardium
EXPERIMENTS WITH THE PRECURSOR OF NORADRENALINE, L–DOPA
EXPERIMENTS WITH ETHYMISOL
EXPERIMENTS WITH THE CENTRAL α-ADRENOBLOCKER, IEM-611
EXPERIMENTS WITH THE GANGLIOBLOCKER, BENZOHEXAMETHONIUM
EXPERIMENTS WITH THE β-BLOCKER, PROPRANOLOL
Chapter 5. The Analysis of Experimental and Clinical Data
Neurogenic Heart Lesions
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