Filling the Gaps in the A&bgr; Cascade Hypothesis of Alzheimer's Disease

Publisher： Bentham Science Publishers

E-ISSN： 1875-5828|3|5|421-430

ISSN： 1567-2050

Source： Current Alzheimer Research, Vol.3, Iss.5, 2006-12, pp. : 421-430

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Cyclic AMP Enhancers and A&bgr; Oligomerization Blockers as Potential Therapeutic Agents in Alzheimer's Disease

By  

Current Alzheimer Research, Vol. 4, Iss. 3, 2007-07 ,pp. : 263-271

Bentham Science Publishers

Access to resources Recommend Favorite

Nogo Receptor Interacts with Brain APP and A&bgr; to Reduce Pathologic Changes in Alzheimer's Transgenic Mice

By  

Current Alzheimer Research, Vol. 4, Iss. 5, 2007-12 ,pp. : 568-570

Bentham Science Publishers

Access to resources Recommend Favorite

Proteolytic Degradation of the Amyloid &bgr;-Protein: The Forgotten Side of Alzheimer's Disease

By  

Current Alzheimer Research, Vol. 3, Iss. 5, 2006-12 ,pp. : 431-435

Bentham Science Publishers

Access to resources Recommend Favorite

Autophagy Enhancer Carbamazepine Alleviates Memory Deficits and Cerebral Amyloid-&bgr; Pathology in a Mouse Model of Alzheimer's Disease

By  

Current Alzheimer Research, Vol. 10, Iss. 4, 2013-05 ,pp. : 433-441

Bentham Science Publishers

Access to resources Recommend Favorite

TGF-&bgr; Pathway as a Potential Target in Neurodegeneration and Alzheimer's

By  

Current Alzheimer Research, Vol. 3, Iss. 3, 2006-07 ,pp. : 191-195

Bentham Science Publishers

Access to resources Recommend Favorite