Ameliorative Effects of GW1929, a Nonthiazolidinedione PPAR&ggr; Agonist,on Inflammation and Apoptosis in Focal Cerebral Ischemic-Reperfusion Injury

Publisher： Bentham Science Publishers

E-ISSN： 1875-5739|8|3|236-245

ISSN： 1567-2026

Source： Current Neurovascular Research, Vol.8, Iss.3, 2011-08, pp. : 236-245

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Preconditioning with Chronic Cerebral Hypoperfusion Reduces a Focal Cerebral Ischemic Injury and Increases Apurinic/Apyrimidinic Endonuclease/Redox Factor-1 and Matrix Metalloproteinase-2 Expression

By  

Current Neurovascular Research, Vol. 4, Iss. 2, 2007-05 ,pp. : 89-97

Bentham Science Publishers

Access to resources Recommend Favorite

The Contribution of Mannose Binding Lectin to Reperfusion Injury after Ischemic Stroke

By  

Current Neurovascular Research, Vol. 8, Iss. 1, 2011-02 ,pp. : 52-63

Bentham Science Publishers

Access to resources Recommend Favorite

Human Mesenchymal Stem Cells Increases Expression of -Tubulin and Angiopoietin 1 and 2 in Focal Cerebral Ischemia and Reperfusion

By  

Current Neurovascular Research, Vol. 10, Iss. 2, 2013-05 ,pp. : 103-111

Bentham Science Publishers

Access to resources Recommend Favorite

Equol is Neuroprotective During Focal Cerebral Ischemia and Reperfusion that Involves p-Src and gp91phox

By  

Current Neurovascular Research, Vol. 11, Iss. 4, 2014-01 ,pp. : 367-377

Bentham Science Publishers

Access to resources Recommend Favorite

Hsp20 Protects Neuroblastoma Cells from Ischemia/Reperfusion Injury by Inhibition of Apoptosis via a Mechanism that Involves the Mitochondrial Pathways

By  

Current Neurovascular Research, Vol. 7, Iss. 4, 2010-11 ,pp. : 281-287

Bentham Science Publishers

Access to resources Recommend Favorite