Decreased cathepsin V expression due to Fli1 deficiency contributes to the development of dermal fibrosis and proliferative vasculopathy in systemic sclerosis

By Noda Shinji   Asano Yoshihide   Takahashi Takehiro   Akamata Kaname   Aozasa Naohiko   Taniguchi Takashi   Ichimura Yohei   Toyama Tetsuo   Sumida Hayakazu   Kuwano Yoshihiro   Yanaba Koichi   Tada Yayoi   Sugaya Makoto   Kadono Takafumi   Sato Shinichi  

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Oxford University Press

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Progranulin Overproduction Due to Fli‐1 Deficiency Contributes to the Resistance of Dermal Fibroblasts to Tumor Necrosis Factor in Systemic Sclerosis

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John Wiley & Sons Inc

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A possible contribution of endothelial CCN1 downregulation due to Fli1 deficiency to the development of digital ulcers in systemic sclerosis

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John Wiley & Sons Inc

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Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin‐Treated Fli‐1–Haploinsufficient Mice

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John Wiley & Sons Inc

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Identification of Cadherin 11 as a Mediator of Dermal Fibrosis and Possible Role in Systemic Sclerosis

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John Wiley & Sons Inc

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