CCN5 overexpression inhibits profibrotic phenotypes via the PI3K/Akt signaling pathway in lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis and in an in vivo model of lung fibrosis

Author：        

Publisher： Spandidos Publications

ISSN： 1107-3756

Source： International Journal of Molecular Medicine, Vol.33, Iss.2, 2014-01, pp. : 478-486

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Related content

CCN5 attenuates profibrotic phenotypes of fibroblasts through the Smad6-CCN2 pathway: Potential role in epidural fibrosis

By              

International Journal of Molecular Medicine, Vol. 36, Iss. 1, 2015-01 ,pp. : 123-129

Spandidos Publications

Access to resources Recommend Favorite

The Number of Interleukin 1 Receptors on Lung Fibroblasts in Patients with Idiopathic Pulmonary Fibrosis

By  

Respiration, Vol. 66, Iss. 3, 1999-06 ,pp. : 236-241

Karger

Access to resources Recommend Favorite

Celecoxib regulates apoptosis and autophagy via the PI3K/Akt signaling pathway in SGC-7901 gastric cancer cells

By                

International Journal of Molecular Medicine, Vol. 33, Iss. 6, 2014-01 ,pp. : 1451-1458

Spandidos Publications

Access to resources Recommend Favorite

CCN1/Cyr61-PI3K/AKT signaling promotes retinal neovascularization in oxygen-induced retinopathy

By        

International Journal of Molecular Medicine, Vol. 36, Iss. 6, 2015-01 ,pp. : 1507-1518

Spandidos Publications

Access to resources Recommend Favorite

The Cardioprotective Effect of Icariin on Ischemia–Reperfusion Injury in Isolated Rat Heart: Potential Involvement of the PI3K‐Akt Signaling Pathway

By  

CARDIOVASCULAR THERAPEUTICS (ELECTRONIC), Vol. 33, Iss. 3, 2015-06 ,pp. : 134-140

John Wiley & Sons Inc

Access to resources Recommend Favorite