The augmentation of O-GlcNAcylation reduces glyoxal-induced cell injury by attenuating oxidative stress in human retinal microvascular endothelial cells

Author：        

Publisher： Spandidos Publications

E-ISSN： 1791-244X|36|4|1019-1027

ISSN： 1107-3756

Source： International Journal of Molecular Medicine, Vol.36, Iss.4, 2015-01, pp. : 1019-1027

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Protein O-GlcNAcylation: A Critical Regulator of the Cellular Response to Stress

By  

Current Signal Transduction Therapy, Vol. 5, Iss. 1, 2010-01 ,pp. : 49-59

Bentham Science Publishers

Access to resources Recommend Favorite

O-GlcNAcylation under hypoxic conditions and its effects on the blood-retinal barrier in diabetic retinopathy

By        

International Journal of Molecular Medicine, Vol. 33, Iss. 3, 2014-01 ,pp. : 624-632

Spandidos Publications

Access to resources Recommend Favorite

Vaccarin attenuates the human EA.hy926 endothelial cell oxidative stress injury through inhibition of Notch signaling

By  

International Journal of Molecular Medicine, Vol. 35, Iss. 1, 2015-01 ,pp. : 135-142

Spandidos Publications

Access to resources Recommend Favorite

Significance of Nitric Oxide and Peroxynitrite in Permeability Changes of the Retinal Microvascular Endothelial Cell Monolayer Induced by Vascular Endothelial Growth Factor

By  

Journal of Vascular Research, Vol. 36, Iss. 6, 1999-12 ,pp. : 510-515

Karger

Access to resources Recommend Favorite

Melissa officials L. extracts protect human retinal pigment epithelial cells against oxidative stress‐induced apoptosis.

By  

ACTA OPHTHALMOLOGICA, Vol. 94, Iss. S256, 2016-10 ,pp. : n/a-n/a

John Wiley & Sons Inc

Access to resources Recommend Favorite