Expression of familial Alzheimer disease presenilin 1 gene attenuates vesicle traffic and reduces peptide secretion in cultured astrocytes devoid of pathologic tissue environment

Publisher： John Wiley & Sons Inc

E-ISSN： 1098-1136|64|2|317-329

ISSN： 0894-1491

Source： GLIA, Vol.64, Iss.2, 2016-02, pp. : 317-329

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Monogenic determinants of familial Alzheimer's disease: presenilin-1 mutations

By Kovacs D. M.   Tanzi R. E.  

Cellular and Molecular Life Sciences CMLS, Vol. 54, Iss. 9, 1998-09 ,pp. : 902-909

Springer Publishing Company

Access to resources Recommend Favorite

Monogenic determinants of familial Alzheimer's disease: presenilin-2 mutations

By Renbaum P.   Levy-Lahad E.  

Cellular and Molecular Life Sciences CMLS, Vol. 54, Iss. 9, 1998-09 ,pp. : 910-919

Springer Publishing Company

Access to resources Recommend Favorite

Alzheimer's disease‐causing proline substitutions lead to presenilin 1 aggregation and malfunction

By  

THE EMBO JOURNAL, Vol. 34, Iss. 22, 2015-11 ,pp. : 2820-2839

John Wiley & Sons Inc

Access to resources Recommend Favorite

GFAP and vimentin deficiency alters gene expression in astrocytes and microglia in wild‐type mice and changes the transcriptional response of reactive glia in mouse model for Alzheimer's disease

By  

GLIA, Vol. 63, Iss. 6, 2015-06 ,pp. : 1036-1056

John Wiley & Sons Inc

Access to resources Recommend Favorite

Ribozyme attenuates reward to morphine in mice by interfering with mper1 gene expression

By Wang Yueqi  

Biological Rhythm Research, Vol. 37, Iss. 2, 2006-04 ,pp. : 157-167

Taylor & Francis Ltd

Access to resources Recommend Favorite