Publisher: Karger
E-ISSN: 1421-9786|9|3|146-151
ISSN: 1015-9770
Source: Cerebrovascular Diseases, Vol.9, Iss.3, 1999-04, pp. : 146-151
Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.
Abstract
Fifty-six subjects with carotid artery disease were assessed by measuring the cerebral blood flow velocity (CBFV) change in response to inhalation of 5% CO2 in air whilst continuously monitoring the blood pressure (BP). Coherent averaging of the data characterised differences in CBFV, BP, resistance area product and critical closing pressure during changes in end-tidal CO2 (ETCO2). The results primarily demonstrate that the augmentation of ETCO2 increases the CBFV and BP, causing a pressure autoregulatory response, and allows the processes of pressure autoregulation and cerebral vascular reserve to be differentiated.
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