NO-Dependent Endothelial Dysfunction in Type II Diabetes Is Aggravated by Dyslipidemia and Hypertension, but Can Be Restored by Angiotensin-Converting Enzyme Inhibition and Weight Loss

Publisher： Karger

E-ISSN： 1423-0135|50|6|486-497

ISSN： 1018-1172

Source： Journal of Vascular Research, Vol.50, Iss.6, 2013-10, pp. : 486-497

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Sarcopenia – A Potential Target for Angiotensin-Converting Enzyme Inhibition?

By  

Gerontology, Vol. 52, Iss. 4, 2006-07 ,pp. : 237-242

Karger

Access to resources Recommend Favorite

Changes in Vascular Distensibility during Angiotensin-Converting Enzyme Inhibition Involve Bradykinin Type 2 Receptors

By  

Journal of Vascular Research, Vol. 41, Iss. 1, 2004-02 ,pp. : 18-27

Karger

Access to resources Recommend Favorite

Role of Angiotensin-Converting Enzyme and Neutral Endopeptidase in Flow-Dependent Remodeling

By  

Journal of Vascular Research, Vol. 41, Iss. 2, 2004-04 ,pp. : 148-156

Karger

Access to resources Recommend Favorite

The Renin-Angiotensin-Aldosterone System: Approaches to Guide Angiotensin-Converting Enzyme Inhibition in Patients with Coronary Artery Disease

By  

Cardiology, Vol. 112, Iss. 4, 2008-10 ,pp. : 303-312

Karger

Access to resources Recommend Favorite

Human Vascular Reactivity and Polymorphisms of the Angiotensin-Converting Enzyme and the Angiotensin Type 1 Receptor Genes

By  

Journal of Vascular Research, Vol. 36, Iss. 6, 1999-12 ,pp. : 445-455

Karger

Access to resources Recommend Favorite