Related content

α-Internexin aggregates are abundant in neuronal intermediate filament inclusion disease (NIFID) but rare in other neurodegenerative diseases

By Cairns Nigel   Uryu Kunihiro   Bigio Eileen   Mackenzie Ian   Gearing Marla   Duyckaerts Charles   Yokoo Hideaki   Nakazato Yoichi   Jaros Evelyn   Perry Robert   Arnold Steven   Lee Virginia   Trojanowski John  

Acta Neuropathologica, Vol. 108, Iss. 3, 2004-09 ,pp. : 213-223

Springer Publishing Company

Access to resources Recommend Favorite

Basophilic inclusion body disease and neuronal intermediate filament inclusion disease: a comparative clinicopathological study

By Yokota Osamu   Tsuchiya Kuniaki   Terada Seishi   Ishizu Hideki   Uchikado Hirotake   Ikeda Manabu   Oyanagi Kiyomitsu   Nakano Imaharu   Murayama Shigeo   Kuroda Shigetoshi   Akiyama Haruhiko  

Acta Neuropathologica, Vol. 115, Iss. 5, 2008-05 ,pp. : 561-575

Springer Publishing Company

Access to resources Recommend Favorite

Spatial patterns of the pathological changes in neuronal intermediate filament inclusion disease (NIFID): an α-internexin immunohistochemical study

By Armstrong R. A.   Cairns N. J.  

Journal of Neural Transmission, Vol. 114, Iss. 4, 2007-04 ,pp. : 451-456

Springer Publishing Company

Access to resources Recommend Favorite

Overexpression of spermidine/spermine N 1 -acetyltransferase elevates the threshold to pentylenetetrazol-induced seizure activity in transgenic mice

By Kaasinen S.K.   Grohn O.H.J.   Keinanen T.A.   Alhonen L.   Janne J.  

Experimental Neurology, Vol. 183, Iss. 2, 2003-10 ,pp. : 645-652

Elsevier

Access to resources Recommend Favorite

Overexpression of human cystatin C in transgenic mice does not affect levels of endogenous brain amyloid β peptide

By Pawlik Monika   Sastre Magdalena   Calero Miguel   Mathews Paul   Schmidt Stephen   Nixon Ralph   Levy Efrat  

Journal of Molecular Neuroscience, Vol. 22, Iss. 1-2, 2004-02 ,pp. : 13-18

Humana Press, Inc

Access to resources Recommend Favorite