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Oxford University Press

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The influence of DNA double-strand break structure on end-joining in human cells

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Characteristics of the end-joining of DNA double-strand breaks by the ataxia-telangiectasia nuclear extract

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The alternative end-joining pathway for repair of DNA double-strand breaks requires PARP1 but is not dependent upon microhomologies

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DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining

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