Determination of α-1 Acid Glycoprotein in Patients with Ph+ Chronic Myeloid Leukemia during the First 13 Weeks of Therapy with STI571

By le Coutre P.   Kreuzer K-A.   Na I-K.   Lupberger J.   Holdhoff M.   Appelt C.   Schwarz M.   Müller C.   Gambacorti-Passerini C.   Platzbecker U.   Bonnet R.   Ehninger G.   Schmidt C.A.  

Blood Cells, Molecules, and Diseases, Vol. 28, Iss. 1, 2002-01 ,pp. : 75-85

Academic Press

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Endoplasmic reticulum stress initiates apoptotic death induced by STI571 inhibition of p210 bcr-abl tyrosine kinase

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Leukemia Research, Vol. 28, Iss. 2, 2004-02 ,pp. : 191-202

Elsevier

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The tyrosine kinase inhibitor STI571, like interferon- , preferentially reduces the capacity for amplification of granulocyte-macrophage progenitors from patients with chronic myeloid leukemia

By Marley S.B.   Deininger M.W.N.   Davidson R.J.   Goldman J.M.   Gordon M.Y.  

Experimental Hematology, Vol. 28, Iss. 5, 2000-05 ,pp. : 551-557

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ABL-kinase domain point mutation as a cause of imatinib (STI571) resistance in CML patient who progress to myeloid blast crisis

By Sacha T.   Hochhaus A.   Hanfstein B.   Muller M.C.   Rudzki Z.   Czopek J.   Wolska-Smolen T.   Czekalska S.   Salamanchuk Z.   Jakobczyk M.   Skotnicki A.B.  

Leukemia Research, Vol. 27, Iss. 12, 2003-12 ,pp. : 1163-1166

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STI571-resistant KT-1 cells are sensitive to interferon- accompanied by the loss of T-cell protein tyrosine phosphatase and prolonged phosphorylation of Stat1

By Shimizu T.   Miyakawa Y.   Oda A.   Kizaki M.   Ikeda Y.  

Experimental Hematology, Vol. 31, Iss. 7, 2003-07 ,pp. : 601-608

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