Huntingtin’s WW domain partners in Huntington’s disease post-mortem brain fulfill genetic criteria for direct involvement in Huntington’s disease pathogenesis

By Passani Lucius A.   Bedford MarkT.  

Human Molecular Genetics, Vol. 9, Iss. 14, 2000-09 ,pp. : 2175-2182

Oxford University Press

Access to resources Recommend Favorite

Aberrant interactions of transcriptional repressor proteins with the Huntington’s disease gene product, huntingtin

By Boutell Jonathan M.   Thomas Philip   Neal James W.   Weston Victoria J.   Duce James   Harper Peter S.   Jones A. Lesley  

Human Molecular Genetics, Vol. 8, Iss. 9, 1999-09 ,pp. : 1647-1655

Oxford University Press

Access to resources Recommend Favorite

Decreased expression of striatal signaling genes in a mouse model of Huntington’s disease

By Luthi-Carter Ruth   Strand Andrew   Peters Nikki L.   Solano Steven M.   Hollingsworth Zane R.   Menon Anil S.   Frey Ariel S.   Spektor Boris S.   Penney Ellen B.   Schilling Gabriele   Ross Christopher A.   Borchelt David R.   Tapscott Stephen J.   Young Anne B.   Cha Jang-Ho J.   Olson James M.  

Human Molecular Genetics, Vol. 9, Iss. 9, 2000-05 ,pp. : 1259-1271

Oxford University Press

Access to resources Recommend Favorite

Neurological abnormalities in a knock-in mouse model of Huntington’s disease

By Detloff P.J.  

Human Molecular Genetics, Vol. 10, Iss. 2, 2001-01 ,pp. : 137-144

Oxford University Press

Access to resources Recommend Favorite

Polyglutamine expansions cause decreased CRE-mediated transcription and early gene expression changes prior to cell death in an inducible cell model of Huntington’s disease

By Schapira A.  

Human Molecular Genetics, Vol. 10, Iss. 17, 2001-08 ,pp. : 1829-1845

Oxford University Press

Access to resources Recommend Favorite