Molecular dissection of ALS-associated toxicity of SOD1 in transgenic mice using an exon-fusion approach

By Deng Han-Xiang   Jiang Hujun   Fu Ronggen   Zhai Hong   Shi Yong   Liu Erdong   Hirano Makito   Dal Canto Mauro C.   Siddique Teepu  

Human Molecular Genetics, Vol. 18, Iss. 3, 2009-02 ,pp. : 594-594

Oxford University Press

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An examination of wild-type SOD1 in modulating the toxicity and aggregation of ALS-associated mutant SOD1

By Prudencio Mercedes   Durazo Armando   Whitelegge Julian P.   Borchelt David R.  

Human Molecular Genetics, Vol. 19, Iss. 24, 2010-12 ,pp. : 4774-4789

Oxford University Press

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Variation in aggregation propensities among ALS-associated variants of SOD1: Correlation to human disease

By Prudencio Mercedes   Hart P. John   Borchelt David R.   Andersen Peter M.  

Human Molecular Genetics, Vol. 18, Iss. 17, 2009-09 ,pp. : 3217-3226

Oxford University Press

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Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common feature

By Wang Jiou  

Human Molecular Genetics, Vol. 12, Iss. 21, 2003-11 ,pp. : 2753-2764

Oxford University Press

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Dismutase-competent SOD1 mutant accumulation in myelinating Schwann cells is not detrimental to normal or transgenic ALS model mice

By Turner Bradley J.   Ackerley Steven   Davies Kay E.   Talbot Kevin  

Human Molecular Genetics, Vol. 19, Iss. 5, 2010-03 ,pp. : 815-824

Oxford University Press

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