DSCR1, overexpressed in Down syndrome, is an inhibitor of calcineurin-mediated signaling pathways

By Fuentes Juan J.   Genescà Lali   Kingsbury Tami J.   Cunningham Kyle W.   Pérez-Riba Mercè   Estivill Xavier   Luna Susana de la  

Human Molecular Genetics, Vol. 9, Iss. 11, 2000-07 ,pp. : 1681-1690

Oxford University Press

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Over-expression of RCAN1 causes Down syndrome-like hippocampal deficits that alter learning and memory

By Martin Katherine R.   Corlett Alicia   Dubach Daphne   Mustafa Tomris   Coleman Harold A.   Parkington Helena C.   Merson Tobias D.   Bourne James A.   Porta Sílvia   Arbonés Maria L.   Finkelstein David I.   Pritchard Melanie A.  

Human Molecular Genetics, Vol. 21, Iss. 13, 2012-07 ,pp. : 3025-3041

Oxford University Press

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Integrative genomics identifies DSCR1 (RCAN1) as a novel NFAT-dependent mediator of phenotypic modulation in vascular smooth muscle cells

By Lee Monica Y.   Garvey Sean M.   Baras Alex S.   Lemmon Julia A.   Gomez Maria F.   Schoppee Bortz Pamela D.   Daum Guenter   LeBoeuf Renee C.   Wamhoff Brian R.  

Human Molecular Genetics, Vol. 19, Iss. 3, 2010-02 ,pp. : 468-479

Oxford University Press

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The DYRK1A gene, encoded in chromosome 21 Down syndrome critical region, bridges between -amyloid production and tau phosphorylation in Alzheimer disease

By Kimura Ryo   Kamino Kouzin   Nuripa Aidaralieva   Kida Tomoyuki   Kazui Hiroaki   Hashimoto Ryota   Tanaka Toshihisa   Kudo Takashi   Yamagata Hidehisa   Tabara Yasuharu   Miki Tetsuro   Akatsu Hiroyasu   Kosaka Kenji   Funakoshi Eishi   Nishitomi Kouhei   Sakaguchi Gaku   Kato Akira   Hattori Hideyuki   Uema Takeshi   Takeda Masatoshi  

Human Molecular Genetics, Vol. 16, Iss. 1, 2007-01 ,pp. : 15-23

Oxford University Press

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Neuroanatomy of Down Syndrome in vivo : A Model of Preclinical Alzheimer’s Disease

By Teipel Stefan   Hampel Harald  

Behavior Genetics, Vol. 36, Iss. 3, 2006-05 ,pp. : 405-415

Springer Publishing Company

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