Kinesin-1 transport reductions enhance human tau hyperphosphorylation, aggregation and neurodegeneration in animal models of tauopathies

By Falzone Toms L.   Gunawardena Shermali   McCleary David   Reis Gerald F.   Goldstein Lawrence S.B.  

Human Molecular Genetics, Vol. 19, Iss. 22, 2010-11 ,pp. : 4399-4408

Oxford University Press

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Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage

By Manczak Maria   Reddy P. Hemachandra  

Human Molecular Genetics, Vol. 21, Iss. 11, 2012-06 ,pp. : 2538-2547

Oxford University Press

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Mitochondrial dysfunction and tau hyperphosphorylation in Ts1Cje, a mouse model for Down syndrome

By Shukkur Ebrahim Abdul   Shimohata Atsushi   Akagi Takumi   Yu Wenxin   Yamaguchi Mika   Murayama Miyuki   Chui Dehua   Takeuchi Tamaki   Amano Kenji   Subramhanya Karthik Harve   Hashikawa Tsutomu   Sago Haruhiko   Epstein Charles J.   Takashima Akihiko   Yamakawa Kazuhiro  

Human Molecular Genetics, Vol. 15, Iss. 18, 2006-09 ,pp. : 2752-2762

Oxford University Press

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Cytoskeleton proteins are modulators of mutant tau-induced neurodegeneration in Drosophila

By Blard Olivier   Feuillette Sébastien   Chaumette Boris   Frébourg Thierry   Campion Dominique   Lecourtois Magalie  

Human Molecular Genetics, Vol. 16, Iss. 5, 2007-03 ,pp. : 555-566

Oxford University Press

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NMNAT suppresses Tau-induced neurodegeneration by promoting clearance of hyperphosphorylated Tau oligomers in a Drosophila model of tauopathy

By Ali Yousuf O.   Ruan Kai   Zhai R. Grace  

Human Molecular Genetics, Vol. 21, Iss. 2, 2012-01 ,pp. : 237-250

Oxford University Press

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