Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protein aggregation

By Witan Heidrun   Kern Andreas   Koziollek-Drechsler Ingrid   Wade Rebecca   Behl Christian   Clement Albrecht M.  

Human Molecular Genetics, Vol. 17, Iss. 10, 2008-05 ,pp. : 1373-1385

Oxford University Press

Access to resources Recommend Favorite

Oxidative stress causes abnormal accumulation of familial amyotrophic lateral sclerosis-related mutant SOD1 in transgenic Caenorhabditis elegans

By Ishii N.   Oeda T.   Shimohama S.   Kitagawa N.   Kohno R.   Imura T.   Shibasaki H.  

Human Molecular Genetics, Vol. 10, Iss. 19, 2001-09 ,pp. : 2013-2023

Oxford University Press

Access to resources Recommend Favorite

The unfolded protein response in familial amyotrophic lateral sclerosis

By Wang Lijun   Popko Brian   Roos Raymond P.  

Human Molecular Genetics, Vol. 20, Iss. 5, 2011-03 ,pp. : 1008-1015

Oxford University Press

Access to resources Recommend Favorite

Mutant FUS proteins that cause amyotrophic lateral sclerosis incorporate into stress granules

By Bosco Daryl A.   Lemay Nathan   Ko Hae Kyung   Zhou Hongru   Burke Chris   Kwiatkowski Thomas J.   Sapp Peter   McKenna-Yasek Diane   Brown Robert H.   Hayward Lawrence J.  

Human Molecular Genetics, Vol. 19, Iss. 21, 2010-11 ,pp. : 4160-4175

Oxford University Press

Access to resources Recommend Favorite

Calcium-permeable AMPA receptors promote misfolding of mutant SOD1 protein and development of amyotrophic lateral sclerosis in a transgenic mouse model

By Tateno Minako  

Human Molecular Genetics, Vol. 13, Iss. 19, 2004-10 ,pp. : 2183-2196

Oxford University Press

Access to resources Recommend Favorite