Regulation of Cardiac Myocyte Contractile Function by Inducible Nitric Oxide Synthase (iNOS): Mechanisms of Contractile Depression by Nitric Oxide

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Journal of Molecular and Cellular Cardiology, Vol. 30, Iss. 2, 1998-02 ,pp. : 303-315

Academic Press

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Exogenous Nitric Oxide Can Trigger a Preconditioned State Through a Free Radical Mechanism, But Endogenous Nitric Oxide Is Not a Trigger of Classical Ischemic Preconditioning

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Inhibition of Angiotensin-converting Enzyme Increases the Nitric Oxide Levels in Canine Ischemic Myocardium

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Cardioprotection by Cyclosporine A in Experimental Ischemia and Reperfusion-Evidence for a Nitric Oxide-dependent Mechanism Mediated by Endothelin

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Effects of inducible nitric oxide synthase (iNOS) deficiency in mice on Sertoli cell proliferation and perinatal testis development

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