Prevention of Autoimmune Diabetes in Nonobese Diabetic Female Mice by Treatment with Recombinant Glutamic Acid Decarboxylase (GAD 65)

By Pleau J-M.   Fernandez-Saravia F.   Esling A.   Homo-Delarche F.   Dardenne M.  

Clinical Immunology, Vol. 76, Iss. 1, 1995-07 ,pp. : 90-95

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High T Cell Responses to the Glutamic Acid Decarboxylase (GAD) Isoform 67 Reflect a Hyperimmune State that Precedes the Onset of Insulin-Dependent Diabetes

By Honeyman M.C.   Stone N.   de Aizpurua H.   Rowley M.J.   Harrison L.C.  

Journal of Autoimmunity, Vol. 10, Iss. 2, 1997-04 ,pp. : 165-173

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Detection of Glutamic Acid Decarboxylase (GAD) Autoantibodies by Indirect Immunofluorescence Using CHO Cells Expressing Recombinant Human GAD65

By Papouchado M.L.   Ermacora M.R.   Poskus E.  

Journal of Autoimmunity, Vol. 9, Iss. 5, 1996-10 ,pp. : 689-697

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Progression to Type 1 Diabetes Is Associated with a Change in the Immunoglobulin Isotype Profile of Autoantibodies to Glutamic Acid Decarboxylase (GAD65)

By Petersen J.S.   Kulmala P.   Clausen J.T.   Knip M.   Dyrberg T.    

Clinical Immunology, Vol. 90, Iss. 2, 1999-02 ,pp. : 276-281

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T Cells with Multiple Fine Specificities are Used by Non-obese Diabetic (NOD) Mice in the Response to GAD(524-543)

By Quinn A.   Sercarz E.E.  

Journal of Autoimmunity, Vol. 9, Iss. 3, 1996-03 ,pp. : 365-370

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