High T Cell Responses to the Glutamic Acid Decarboxylase (GAD) Isoform 67 Reflect a Hyperimmune State that Precedes the Onset of Insulin-Dependent Diabetes

By Honeyman M.C.   Stone N.   de Aizpurua H.   Rowley M.J.   Harrison L.C.  

Journal of Autoimmunity, Vol. 10, Iss. 2, 1997-04 ,pp. : 165-173

Academic Press

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Clinical Evaluation of Non-Insulin-Dependent Diabetes Mellitus Patients with Autoantibodies to Glutamic Acid Decarboxylase

By Abiru N.   Takino H.   Yano M.   Kawasaki E.   Yamasaki H.   Yamaguchi Y.   Akazawa S.   Nagataki S.  

Journal of Autoimmunity, Vol. 9, Iss. 5, 1996-10 ,pp. : 683-688

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Antibody to the Mr 65,000 Isoform of Glutamic Acid Decarboxylase are Detected in Non-Insulin-Dependent Diabetes in Japanese

By Kasuga A.   Maruyama T.   Ozawa Y.   Takei I.   Falorni A.   Lernmark A.   Saruta T.  

Journal of Autoimmunity, Vol. 9, Iss. 1, 1996-01 ,pp. : 105-111

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IA-2 Antibodies Are Only Positive in Association With GAD 65 and Islet Cell Antibodies in Islet Transplanted Insulin-Dependent Diabetes Mellitus Patients

By Jaeger C.   Brendel M.D.   Hering B.J.   Eckhard M.   Federlin K.   Bretzel R.G.  

Transplantation Proceedings, Vol. 30, Iss. 2, 1998-03 ,pp. : 659-660

Elsevier

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Progression to Type 1 Diabetes Is Associated with a Change in the Immunoglobulin Isotype Profile of Autoantibodies to Glutamic Acid Decarboxylase (GAD65)

By Petersen J.S.   Kulmala P.   Clausen J.T.   Knip M.   Dyrberg T.    

Clinical Immunology, Vol. 90, Iss. 2, 1999-02 ,pp. : 276-281

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