The curly-tail (ct) mouse, an animal model of neural tube defects, displays altered homocysteine metabolism without folate responsiveness or a defect in Mthfr

Author: Tran P.   Hiou-Tim F.   Frosst P.   Lussier-Cacan S.   Bagley P.   Selhub J.   Bottiglieri T.   Rozen R.  

Publisher: Academic Press

ISSN: 1096-7192

Source: Molecular Genetics and Metabolism, Vol.76, Iss.4, 2002-08, pp. : 297-304

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content