Author: Saito Fumio Imai Shinobu Tanaka Nobuyuki Tanaka Hideyuki Suzuki Kazutaka Takase Hidehito Aoyama Hiroshi Matsudaira Kagari Ebuchi Tomonori Akamine Yasuyuki Takahashi Naoyuki Sugino Keiichi Kanmatsuse Katsuo Yagi Hiroshi Kushiro Toshio
Publisher: Informa Healthcare
ISSN: 1064-1963
Source: Clinical and Experimental Hypertension, Vol.29, Iss.3, 2007-03, pp. : 165-173
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Abstract
Basic autonomic nervous function was evaluated in patients with neurocardiogenic syncope (NCS). Atropine, isoproterenol, propranolol, phenylephrine, and phentolamine were administered successively, and parasympathetic nerve activity and β- (and α-) activity, sensitivity, and secretion of the sympathetic nerve were determined in patients with NCS and control subjects. In patients with NCS, β- and α- sensitivity were higher and β-activity and β- and α-secretion lower than in control subjects. In patients with NCS, the increased basic β-sensitivity may contribute to induce strong cardiac contractions and augment ventricular mechanoreceptor response, and a compensatory state against diminished neuronal sympathetic activity is suggested by the increased α-sensitivity.
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