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Increased γ-H2AX by exposure to a 60-Hz magnetic fields combined with ionizing radiation, but not hydrogen peroxide, in non-tumorigenic human cell lines

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Decay of γ-H2AX foci correlates with potentially lethal damage repair in prostate cancer cells

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Fully automated interpretation of ionizing radiation-induced γH2AX foci by the novel pattern recognition system AKLIDES^®

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Kinetics and dose-response of residual 53BP1/γ-H2AX foci: Co-localization, relationship with DSB repair and clonogenic survival

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