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Oxford University Press

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The RAD5 Gene Product Is Involved in the Avoidance of Non-Homologous End-Joining of DNA Double Strand Breaks in the Yeast Saccharomyces Cerevisiae

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Competing roles of DNA end resection and non-homologous end joining functions in the repair of replication-born double-strand breaks by sister-chromatid recombination

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Effects of camptothecin on double-strand break repair by non-homologous end-joining in DNA mismatch repair-deficient human colorectal cancer cell lines

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The alternative end-joining pathway for repair of DNA double-strand breaks requires PARP1 but is not dependent upon microhomologies

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