Defects in mTR stability and telomerase activity produced by the Dkc1 A353V mutation in dyskeratosis congenita are rescued by a peptide from the dyskerin TruB domain

Author: Machado-Pinilla Rosario  

Publisher: Springer Publishing Company

ISSN: 1699-048X

Source: Clinical and Translational Oncology, Vol.14, Iss.10, 2012-10, pp. : 755-763

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