Eliminating membrane depolarization caused by the Alzheimer peptide Aβ(1–42, aggr.)

By Blanchard B.J.   Stockwell B.R.   Ingram V.M.  

Biochemical and Biophysical Research Communications, Vol. 293, Iss. 4, 2002-05 ,pp. : 1204-1208

Elsevier

Access to resources Recommend Favorite

NMR and CD studies on the interaction of Alzheimer β-amyloid peptide (12–28) with β-cyclodextrin

By Qin X-r.   Abe H.   Nakanishi H.  

Biochemical and Biophysical Research Communications, Vol. 297, Iss. 4, 2002-10 ,pp. : 1011-1015

Elsevier

Access to resources Recommend Favorite

Synthesis, aggregation, neurotoxicity, and secondary structure of various Aβ1–42 mutants of familial Alzheimer's disease at positions 21–23

By Murakami K.   Irie K.   Morimoto A.   Ohigashi H.   Shindo M.   Nagao M.   Shimizu T.   Shirasawa T.  

Biochemical and Biophysical Research Communications, Vol. 294, Iss. 1, 2002-05 ,pp. : 5-10

Elsevier

Access to resources Recommend Favorite

4,4^′-Dianilino-1,1^′-binaphthyl-5,5^′-disulfonate: report on non-β-sheet conformers of Alzheimer's peptide β(1–40)

By LeVine III H.  

Archives of Biochemistry and Biophysics, Vol. 404, Iss. 1, 2002-08 ,pp. : 106-115

Elsevier

Access to resources Recommend Favorite

Amyloid β-peptide (1-42)-induced Oxidative Stress and Neurotoxicity: Implications for Neurodegeneration in Alzheimer's Disease Brain. A Review

By Allan Butterfield D.  

Free Radical Research, Vol. 36, Iss. 12, 2002-01 ,pp. : 1307-1313

Informa Healthcare

Access to resources Recommend Favorite