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Informa Healthcare

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Mitochondrial Dysfunction in the Senescence Accelerated Mouse (SAM)

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Proteomic analysis for protein carbonyl as an indicator of oxidative damage in senescence-accelerated mice

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Thermolabile 8-hydroxyguanine DNA glycosylase with low activity in senescence-accelerated mice due to a single-base mutation - roles of FNR, ArcA, and Fur

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Glutamate receptors regulate the level of reactive oxygen species in neurons of senescence accelerated mice (SAM) strain

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