Profile for Amyloid-β and Tau Expression in Primary Cortical Cultures from 3xTg-AD Mice

Author： Vale Carmen   Alonso Eva   Rubiolo Juan   Vieytes Mercedes   LaFerla Frank   Giménez-Llort Lydia   Botana Luis  

Publisher： Springer Publishing Company

ISSN： 0272-4340

Source： Cellular and Molecular Neurobiology, Vol.30, Iss.4, 2010-05, pp. : 577-590

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

What can we learn from regional vulnerability to amyloid-β accumulation in nondemented individuals?

By Shinohara Mitsuru   Bu Guojun  

Neurodegenerative Disease Management, Vol. 3, Iss. 3, 2013-06 ,pp. : 187-189

Future Medicine

Access to resources Recommend Favorite

Intravenous Immunoglobulin Reduces Tau Pathology and Preserves Neuroplastic Gene Expression in the 3xTg Mouse Model of Alzheimer’s Disease

By  

Current Alzheimer Research, Vol. 11, Iss. 7, 2014-01 ,pp. : 655-663

Bentham Science Publishers

Access to resources Recommend Favorite

Mitochondria: The Common Upstream Driver of Amyloid-&bgr; and Tau Pathology in Alzheimer's Disease

By  

Current Alzheimer Research, Vol. 8, Iss. 5, 2011-08 ,pp. : 563-572

Bentham Science Publishers

Access to resources Recommend Favorite

β-Amyloid Fragment 25–35 Causes Mitochondrial Dysfunction in Primary Cortical Neurons

By Casley C.S.   Land J.M.   Sharpe M.A.   Clark J.B.   Duchen M.R.   Canevari L.  

Neurobiology of Disease, Vol. 10, Iss. 3, 2002-08 ,pp. : 258-267

Academic Press

Access to resources Recommend Favorite

Intraneuronal tau aggregation precedes diffuse plaque deposition, but amyloid-β changes occur before increases of tau in cerebrospinal fluid

By Braak Heiko   Zetterberg Henrik   Tredici Kelly   Blennow Kaj  

Acta Neuropathologica, Vol. 126, Iss. 5, 2013-11 ,pp. : 631-641

Springer Publishing Company

Access to resources Recommend Favorite