Role of Integrins in Cell Adhesion and Polarity in Normal Keratinocytes and Human Skin Pathologies

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THE JOURNAL OF DERMATOLOGY, Vol. 21, Iss. 11, 1994-11 ,pp. : 821-828

John Wiley & Sons Inc

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Par‐aPKC‐dependent and ‐independent mechanisms cooperatively control cell polarity, Hippo signaling, and cell positioning in 16‐cell stage mouse embryos

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DEVELOPMENT, GROWTH & DIFFERENTIATION (ELECTRONIC), Vol. 57, Iss. 8, 2015-10 ,pp. : 544-556

John Wiley & Sons Inc

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The Planar Cell Polarity Transmembrane Protein Vangl2 Promotes Dendrite, Spine and Glutamatergic Synapse Formation in the Mammalian Forebrain

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Molecular Neuropsychiatry, Vol. 2, Iss. 2, 2016-06 ,pp. : 107-114

Karger

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The Organization of Tight Junctions in Epithelia: Implications for Mammary Gland Biology and Breast Tumorigenesis: Cell Adhesion in Mammary Gland Biology and Neoplasia

By Itoh Masahiko   Bissell Mina J.  

Journal of Mammary Gland Biology and Neoplasia, Vol. 8, Iss. 4, 2003-10 ,pp. : 449-462

Springer Publishing Company

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Cover Photograph: Hippo signaling controls cell fates in preimplantation mouse embryos. Slightly different mechanisms operate in regulation of Hippo signaling between 16‐cell stage and early blastocyst stage with more than 32 cells. Panels show the confocal images of the 16‐cell (left panel) and early blastocyst (right panel) stage embryos stained with phosphorylated Ezrin/Radixin/Moesin (magenta), a marker for apical domain and cell polarization, and the Hippo pathway coactivator Yap (green). In the early blastocysts, cell position regulates cell polarization through the Par‐aPKC system, and cell polarity controls Hippo signaling. Therefore, all the outer cells are polarized and exhibit nuclear Yap. In contrast, at the 16‐cell stage, Par‐aPKC‐independent mechanism and asymmetric cell division are also involved in regulation of cell polarization. Therefore, some outer cells are apolar and show cytoplasmic Yap.

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DEVELOPMENT, GROWTH & DIFFERENTIATION (ELECTRONIC), Vol. 57, Iss. 8, 2015-10 ,pp. : i-i

John Wiley & Sons Inc

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