Sustained glucose-stimulated insulin secretion in mouse islets is not culture-dependent

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Springer Publishing Company

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Depletion of the catalytic subunit of protein phosphatase-2A (PP2Ac) markedly attenuates glucose-stimulated insulin secretion in pancreatic β-cells

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Exendin 4 controls insulin production in rat islet beta cells predominantly by potentiation of glucose-stimulated proinsulin biosynthesis at the translational level

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Increasing uncoupling protein-2 in pancreatic beta cells does not alter glucose-induced insulin secretion but decreases production of reactive oxygen species

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Oscillations of sub-membrane ATP in glucose-stimulated beta cells depend on negative feedback from Ca²⁺

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