Increment of HFABP Level in Coronary Artery In-Stent Restenosis Segments in Diabetic and Nondiabetic Minipigs: HFABP Overexpression Promotes Multiple Pathway-Related Inflammation, Growth and Migration in Human Vascular Smooth Muscle Cells

Publisher： Karger

E-ISSN： 1423-0135|53|1-2|27-38

ISSN： 1018-1172

Source： Journal of Vascular Research, Vol.53, Iss.1-2, 2016-07, pp. : 27-38

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Stainless Steel Ions Stimulate Increased Thrombospondin-1-Dependent TGF-Beta Activation by Vascular Smooth Muscle Cells: Implications for In-Stent Restenosis

By  

Journal of Vascular Research, Vol. 47, Iss. 4, 2009-12 ,pp. : 309-322

Karger

Access to resources Recommend Favorite

Different Migration of Vascular Smooth Muscle Cells from Human Coronary Artery Bypass Vessels

By  

Journal of Vascular Research, Vol. 44, Iss. 2, 2007-01 ,pp. : 149-156

Karger

Access to resources Recommend Favorite

Non-Bone Marrow Origin of Neointimal Smooth Muscle Cells in Experimental In-Stent Restenosis in Rats

By  

Journal of Vascular Research, Vol. 45, Iss. 6, 2008-04 ,pp. : 493-502

Karger

Access to resources Recommend Favorite

Pathogen Burden, Inflammation, Proliferation and Apoptosis in Human In-Stent Restenosis

By  

Journal of Vascular Research, Vol. 41, Iss. 6, 2004-12 ,pp. : 525-534

Karger

Access to resources Recommend Favorite

Association of RAGE Gene Polymorphisms with In-Stent Restenosis in Non-Diabetic Korean Population

By  

Cardiology, Vol. 107, Iss. 4, 2006-09 ,pp. : 261-268

Karger

Access to resources Recommend Favorite