Kinesin Expression in the Central Nervous System of Humans and Transgenic hSOD1G93A Mice with Amyotrophic Lateral Sclerosis

Publisher： Karger

E-ISSN： 1660-2862|12|2|71-80

ISSN： 1660-2854

Source： Neurodegenerative Diseases, Vol.12, Iss.2, 2012-09, pp. : 71-80

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Modeling Amyotrophic Lateral Sclerosis in hSOD1G93A Transgenic Swine

By  

Neurodegenerative Diseases, Vol. 13, Iss. 4, 2013-10 ,pp. : 246-254

Karger

Access to resources Recommend Favorite

Prevention of Motor Neuron Degeneration by Novel Iron Chelators in SOD1G93A Transgenic Mice of Amyotrophic Lateral Sclerosis

By  

Neurodegenerative Diseases, Vol. 8, Iss. 5, 2011-02 ,pp. : 310-321

Karger

Access to resources Recommend Favorite

An Astrocyte Regenerative Response from Vimentin-Containing Cells in the Spinal Cord of Amyotrophic Lateral Sclerosis's Disease-Like Transgenic (G93A SOD1) Mice

By  

Neurodegenerative Diseases, Vol. 15, Iss. 1, 2015-01 ,pp. : 1-12

Karger

Access to resources Recommend Favorite

Mutant SOD1 Instability: Implications for Toxicity in Amyotrophic Lateral Sclerosis

By  

Neurodegenerative Diseases, Vol. 2, Iss. 3-4, 2006-01 ,pp. : 115-127

Karger

Access to resources Recommend Favorite

SOD-1 inhibits FAS expression in cortex of APP transgenic mice

By Chen Z.  

Apoptosis, Vol. 10, Iss. 3, 2005-05 ,pp. : 499-502

Springer Publishing Company

Access to resources Recommend Favorite