Tumor Necrosis Factor and Motoneuronal Degeneration: An Open Problem

Publisher： Karger

E-ISSN： 1423-0216|9|4|178-182

ISSN： 1021-7401

Source： Neuroimmunomodulation, Vol.9, Iss.4, 2002-02, pp. : 178-182

Disclaimer: Any content in publications that violate the sovereignty, the constitution or regulations of the PRC is not accepted or approved by CNPIEC.

Previous Menu Next

Abstract

Related content

Interferon β-1a Induces Tumor Necrosis Factor Receptor 1 but Decreases Tumor Necrosis Factor Receptor 2 Leukocyte mRNA Levels in Relapsing-Remitting Multiple Sclerosis

By  

Neuroimmunomodulation, Vol. 16, Iss. 3, 2009-02 ,pp. : 171-176

Karger

Access to resources Recommend Favorite

Corticosterone Changes in Response to Stressors, Acute and Protracted Actions of Tumor Necrosis Factor-α, and Lipopolysaccharide Treatments in Mice Lacking the Tumor Necrosis Factor-α p55 Receptor Gene

By  

Neuroimmunomodulation, Vol. 11, Iss. 4, 2004-07 ,pp. : 241-246

Karger

Access to resources Recommend Favorite

Lipopolysaccharide- and Tumor Necrosis Factor-α-Induced Changes in Prolactin Secretion and Dopaminergic Activity in the Hypothalamic-Pituitary Axis

By  

Neuroimmunomodulation, Vol. 10, Iss. 1, 2002-08 ,pp. : 30-39

Karger

Access to resources Recommend Favorite

Lipopolysaccharide-Induced Tumor Necrosis Factor-Alpha Release Is Controlled by the Central Nervous System

By  

Neuroimmunomodulation, Vol. 9, Iss. 3, 2001-12 ,pp. : 148-156

Karger

Access to resources Recommend Favorite

Exhaustive Exercise Reduces Tumor Necrosis Factor-α Production in Response to Lipopolysaccharide in Mice

By  

Neuroimmunomodulation, Vol. 17, Iss. 4, 2010-03 ,pp. : 279-286

Karger

Access to resources Recommend Favorite