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Toxicology, Vol. 165, Iss. 1, 2001-08 ,pp. : 51-63

Elsevier

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Mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced decrease in anti-CD3-activated CD4 + T cells: the roles of apoptosis, Fas, and TNF

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Toxicology, Vol. 170, Iss. 1, 2002-01 ,pp. : 139-151

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Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis——Persistent pollutants affect programmed cell death

By Chopra Martin   Schrenk Dieter  

Critical Reviews in Toxicology, Vol. 41, Iss. 4, 2011-04 ,pp. : 292-320

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Role of Fas apoptosis and MHC genes in 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced immunotoxicity of T cells

By Rhile M.J.   Nagarkatti M.   Nagarkatti P.S.  

Toxicology, Vol. 110, Iss. 1, 1996-06 ,pp. : 153-167

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T Lymphocytes Are Direct, Aryl Hydrocarbon Receptor (AhR)-Dependent Targets of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD): AhR Expression in Both CD4⁺ and CD8⁺ T Cells Is Necessary for Full Suppression of a Cytotoxic T Lymphocyte Response by TCDD

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