Amyloid Phenotype Characterization of Transgenic Mice Overexpressing both Mutant Amyloid Precursor Protein and Mutant Presenilin 1 Transgenes

By McGowan E.   Sanders S.   Iwatsubo T.   Takeuchi A.   Saido T.   Zehr C.   Yu X.   Uljon S.   Wang R.   Mann D.   Dickson D.   Duff K.  

Neurobiology of Disease, Vol. 6, Iss. 4, 1999-08 ,pp. : 231-244

Academic Press

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Neurodegenerative Changes Associated with -Amyloid Deposition in the Brains of Mice Carrying Mutant Amyloid Precursor Protein and Mutant Presenilin-1 Transgenes

By Kurt M.A.   Davies D.C.   Kidd M.   Duff K.   Rolph S.C.   Jennings K.H.   Howlett D.R.  

Experimental Neurology, Vol. 171, Iss. 1, 2001-09 ,pp. : 59-71

Elsevier

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Enhanced Synaptic Potentiation in Transgenic Mice Expressing presenilin 1 Familial Alzheimer's Disease Mutation Is Normalized with a Benzodiazepine

By Zaman S.H.   Parent A.   Laskey A.   Lee M.K.   Borchelt D.R.   Sisodia S.S.   Malinow R.  

Neurobiology of Disease, Vol. 7, Iss. 1, 2000-02 ,pp. : 54-63

Academic Press

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Synaptic Transmission and Hippocampal Long-Term Potentiation in Transgenic Mice Expressing FAD-Linked Presenilin 1

By Parent A.   Linden D.J.   Sisodia S.S.   Borchelt D.R.  

Neurobiology of Disease, Vol. 6, Iss. 1, 1999-02 ,pp. : 56-62

Academic Press

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Presenilin-1 Mutation Sensitizes Oligodendrocytes to Glutamate and Amyloid Toxicities, and Exacerbates White Matter Damage and Memory Impairment in Mice

By Pak Kirk   Chan Sic L.   Mattson Mark P.  

NeuroMolecular Medicine, Vol. 3, Iss. 1, 2003-02 ,pp. : 53-64

Humana Press, Inc

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